Toxicological effects of fine particulate matter derived from the destruction of the World Trade Center (Perfect)
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| Preise | 2011 | 2013 | 2014 |
|---|---|---|---|
| Schnitt | € 10,00 | € 66,82 | € 85,66 |
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Deutsche Kolonialzeitung (17 )
EN PB NWISBN: 9781234864088 bzw. 1234864088, in Englisch, Rarebooksclub.com, Taschenbuch, neu.
Von Händler/Antiquariat, BuySomeBooks [52360437], Las Vegas, NV, U.S.A.
This item is printed on demand. Paperback. 64 pages. OCLC Number: 52707258 Excerpt: . . . greater sensitivity to agents which constrict breathing passages ) comparable to NIST 1649a and to a greater degree than ROFA. Doses of 10 and 32 g administered directly into the airways, or 3 inhalation at 10 mg m, did not induce significant inflammation or hyperresponsiveness. The significant degree of airway hyperresponsiveness induced by the high dose of WTC PM implies 2. 5 that components of the dust can promote mechanisms of airway obstruction. The results from these studies indicate that a high dose of WTC dust as PM would be 2. 5 necessary to elicit effects in healthy people. Hypothetical calculations are presented indicating that 3 a healthy worker at Ground Zero would have to inhale about 425 g m WTC PM for 8 hours to 2. 5 achieve the same dose per tracheobronchial surface area as occurred with the high dose of WTC PM used in the mouse studies. These high concentrations are conceivable in the aftermath of the 2. 5 collapse of the towers when rescue and salvage efforts were in effect. Therefore a healthy worker without respiratory protection could have inhaled enough WTC PM to cause pulmonary 2. 5 inflammation, airway hyperresponsiveness, and manifestations of sensory irritation such as cough. Species differences in responses to inhalation of WTC PM are unknown and were not considered 2. 5 in these calculations. Individuals who are especially sensitive to inhalation of dusts, such as asthmatics, may experience these effects at lower doses of inhaled WTC PM. These studies 2. 5 suggest that most healthy people would not respond to a single exposure to moderately high WTC 3 PM levels ( about 130 g m or less for 8 hours ) with any adverse respiratory responses. However, 2. 5 it should be emphasized that the effects of chronic ( long-term ) or repeated exposures to lower levels of WTC PM, or the persistence of any respiratory effects are unknown and were not components 2. 5 of this study. Although only fine PM was tested. . . This item ships from La Vergne,TN.
This item is printed on demand. Paperback. 64 pages. OCLC Number: 52707258 Excerpt: . . . greater sensitivity to agents which constrict breathing passages ) comparable to NIST 1649a and to a greater degree than ROFA. Doses of 10 and 32 g administered directly into the airways, or 3 inhalation at 10 mg m, did not induce significant inflammation or hyperresponsiveness. The significant degree of airway hyperresponsiveness induced by the high dose of WTC PM implies 2. 5 that components of the dust can promote mechanisms of airway obstruction. The results from these studies indicate that a high dose of WTC dust as PM would be 2. 5 necessary to elicit effects in healthy people. Hypothetical calculations are presented indicating that 3 a healthy worker at Ground Zero would have to inhale about 425 g m WTC PM for 8 hours to 2. 5 achieve the same dose per tracheobronchial surface area as occurred with the high dose of WTC PM used in the mouse studies. These high concentrations are conceivable in the aftermath of the 2. 5 collapse of the towers when rescue and salvage efforts were in effect. Therefore a healthy worker without respiratory protection could have inhaled enough WTC PM to cause pulmonary 2. 5 inflammation, airway hyperresponsiveness, and manifestations of sensory irritation such as cough. Species differences in responses to inhalation of WTC PM are unknown and were not considered 2. 5 in these calculations. Individuals who are especially sensitive to inhalation of dusts, such as asthmatics, may experience these effects at lower doses of inhaled WTC PM. These studies 2. 5 suggest that most healthy people would not respond to a single exposure to moderately high WTC 3 PM levels ( about 130 g m or less for 8 hours ) with any adverse respiratory responses. However, 2. 5 it should be emphasized that the effects of chronic ( long-term ) or repeated exposures to lower levels of WTC PM, or the persistence of any respiratory effects are unknown and were not components 2. 5 of this study. Although only fine PM was tested. . . This item ships from La Vergne,TN.
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Toxicological effects of fine particulate matter derived from the destruction of the World Trade Center
EN PB NWISBN: 9781234864088 bzw. 1234864088, in Englisch, Taschenbuch, neu.
Von Händler/Antiquariat, BuySomeBooks [52360437], Las Vegas, NV, U.S.A.
This item is printed on demand. Paperback. OCLC Number: 52707258 Excerpt: . . . greater sensitivity to agents which constrict breathing passages ) comparable to NIST 1649a and to a greater degree than ROFA. Doses of 10 and 32 g administered directly into the airways, or 3 inhalation at 10 mg m, did not induce significant inflammation or hyperresponsiveness. The significant degree of airway hyperresponsiveness induced by the high dose of WTC PM implies 2. 5 that components of the dust can promote mechanisms of airway obstruction. The results from these studies indicate that a high dose of WTC dust as PM would be 2. 5 necessary to elicit effects in healthy people. Hypothetical calculations are presented indicating that 3 a healthy worker at Ground Zero would have to inhale about 425 g m WTC PM for 8 hours to 2. 5 achieve the same dose per tracheobronchial surface area as occurred with the high dose of WTC PM used in the mouse studies. These high concentrations are conceivable in the aftermath of the 2. 5 collapse of the towers when rescue and salvage efforts were in effect. Therefore a healthy worker without respiratory protection could have inhaled enough WTC PM to cause pulmonary 2. 5 inflammation, airway hyperresponsiveness, and manifestations of sensory irritation such as cough. Species differences in responses to inhalation of WTC PM are unknown and were not considered 2. 5 in these calculations. Individuals who are especially sensitive to inhalation of dusts, such as asthmatics, may experience these effects at lower doses of inhaled WTC PM. These studies 2. 5 suggest that most healthy people would not respond to a single exposure to moderately high WTC 3 PM levels ( about 130 g m or less for 8 hours ) with any adverse respiratory responses. However, 2. 5 it should be emphasized that the effects of chronic ( long-term ) or repeated exposures to lower levels of WTC PM, or the persistence of any respiratory effects are unknown and were not components 2. 5 of this study. Although only fine PM was tested. . . This item ships from La Vergne,TN.
This item is printed on demand. Paperback. OCLC Number: 52707258 Excerpt: . . . greater sensitivity to agents which constrict breathing passages ) comparable to NIST 1649a and to a greater degree than ROFA. Doses of 10 and 32 g administered directly into the airways, or 3 inhalation at 10 mg m, did not induce significant inflammation or hyperresponsiveness. The significant degree of airway hyperresponsiveness induced by the high dose of WTC PM implies 2. 5 that components of the dust can promote mechanisms of airway obstruction. The results from these studies indicate that a high dose of WTC dust as PM would be 2. 5 necessary to elicit effects in healthy people. Hypothetical calculations are presented indicating that 3 a healthy worker at Ground Zero would have to inhale about 425 g m WTC PM for 8 hours to 2. 5 achieve the same dose per tracheobronchial surface area as occurred with the high dose of WTC PM used in the mouse studies. These high concentrations are conceivable in the aftermath of the 2. 5 collapse of the towers when rescue and salvage efforts were in effect. Therefore a healthy worker without respiratory protection could have inhaled enough WTC PM to cause pulmonary 2. 5 inflammation, airway hyperresponsiveness, and manifestations of sensory irritation such as cough. Species differences in responses to inhalation of WTC PM are unknown and were not considered 2. 5 in these calculations. Individuals who are especially sensitive to inhalation of dusts, such as asthmatics, may experience these effects at lower doses of inhaled WTC PM. These studies 2. 5 suggest that most healthy people would not respond to a single exposure to moderately high WTC 3 PM levels ( about 130 g m or less for 8 hours ) with any adverse respiratory responses. However, 2. 5 it should be emphasized that the effects of chronic ( long-term ) or repeated exposures to lower levels of WTC PM, or the persistence of any respiratory effects are unknown and were not components 2. 5 of this study. Although only fine PM was tested. . . This item ships from La Vergne,TN.
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Toxicological effects of fine particulate matter derived from the destruction of the World Trade Center (Perfect)
EN NWISBN: 9781234864088 bzw. 1234864088, in Englisch, neu.
Von Händler/Antiquariat, ABC Books [9235530], Lowfield Heath, CRAWL, United Kingdom.
Perfect. OCLC Number: 52707258 Excerpt: .greater sensitivity to agents which constrict breathing passages ) comparable to NIST 1649a and to a greater degree than ROFA. Doses O.Shipping may be from our UK, US or Australian warehouse depending on stock availability. This item is printed on demand. 64 pages. 0.136.
Perfect. OCLC Number: 52707258 Excerpt: .greater sensitivity to agents which constrict breathing passages ) comparable to NIST 1649a and to a greater degree than ROFA. Doses O.Shipping may be from our UK, US or Australian warehouse depending on stock availability. This item is printed on demand. 64 pages. 0.136.
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Toxicological Effects of Fine Particulate Matter Derived from the Destruction of the World Trade Center (2013)
EN NWISBN: 9781234864088 bzw. 1234864088, in Englisch, General Books LLC, General Books LLC, General Books LLC, neu.
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Stephen H. Gavett National Health and,Paperback, English-language edition,Pub by General Books LLC.
Stephen H. Gavett National Health and,Paperback, English-language edition,Pub by General Books LLC.
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